Reaction: HCO3- transport through ion channel

- in pathway: ABC-family proteins mediated transport
Regulation of epithelial chloride flux, which is defective in patients with cystic fibrosis, may be mediated by phosphorylation of the cystic fibrosis transmembrane conductance regulator (CFTR) by cyclic AMP-dependent protein kinase (PKA) or protein kinase C (PKC). CFTR regulates both HCO(3)(-) secretion and HCO(3)(-) salvage in secretory epithelia.
Reaction - small molecule participants:
Pi [cytosol]
ADP [cytosol]
HCO3- [extracellular region]
H2O [cytosol]
HCO3- [cytosol]
ATP [cytosol]
Reactome.org reaction link: R-HSA-383190

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Reaction input - small molecules:
water
ChEBI:15377
hydrogencarbonate
ChEBI:17544
ATP(4-)
ChEBI:30616
Reaction output - small molecules:
hydrogenphosphate
ChEBI:43474
ADP(3-)
ChEBI:456216
hydrogencarbonate
ChEBI:17544
Reactome.org link: R-HSA-383190